Office Heat - Erotic Short Story for Women
She was His for the taking. Right now I have another one in mind," He said, reaching his hand underneath and between her legs and inserting His fingers into her pussy, which was now leaking her cum and as wet as could be. Indeed He had never seen one wetter. It was time. Master Leon released her, stepped back, unbuckled his belt and undid his trousers.
They dropped to the floor. His boxers looked like a tent, with His mighty shaft the tent pole. He pulled them down next, exposing His fully hard, throbbing man meat. All the while He kept his gaze on His target. Janice was fresh meat, and now He intended to devour her. Leon leaned forward, placing His cock between her cheeks, extending onto her back. Leaning back, he traced the head of His cock along her crack, then underneath, right up to her glistening cunt. He knew the next part could cause more noise than He cared for, even though the day was nearly over and many were leaving the office.
So He reached across to her mouth, covering it with one hand, placed the other hand on her hip, and dove into her swampy pussy with His African breeding stick. He felt a wave of pleasure as her warm pussy engulfed His rock hard cock. He only went half way in so as not to overwhelm her any more than she was already. He stroked her short brown hair repeatedly as one does a pet animal such as a dog or cat. As Master Leon well knew, this reassures and comforts livestock, letting it know that it is in trusted, loving hands.
Sure enough, in His expert control, Janice relaxed once again. Now you are adjusting to serving Me. This time she only squealed into his hand. Do you understand? After a moment Janice, through a haze of pleasure, answered, "Yes. She will be so easily trained.
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With that he began thrusting out and back into her dripping cunt. When she grunted, He slowed down and reminded her, "Quiet!
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I want you to only hear My voice," before picking up the pace again. It was all Janice could do to keep from yelling out in ecstasy but she controlled herself, letting only tiny gasps escape. Master Leon coached her with praise all along the way. He leaned down to her ear and whispered:.
He gave her a moment to compose herself. She was after all getting the fucking of a lifetime, and maybe her first fucking at all! Through her heavy panting she managed:. Master Leon began fucking her again with long, deep strokes. Damn right!
I own that pussy. Got that? Say it.
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Tell me who it belongs to! Other measures, such as leukocyte vitamin C concentration, could be more accurate indicators of tissue vitamin C levels, but they are more difficult to assess and the results are not always reliable [ 4 , 9 , 16 ]. Acute vitamin C deficiency leads to scurvy [ 7 , 8 , 11 ]. Initial symptoms can include fatigue probably the result of impaired carnitine biosynthesis , malaise, and inflammation of the gums [ 4 , 11 ].
As vitamin C deficiency progresses, collagen synthesis becomes impaired and connective tissues become weakened, causing petechiae, ecchymoses, purpura, joint pain, poor wound healing, hyperkeratosis, and corkscrew hairs [ 1 , 2 , 4 , ]. Additional signs of scurvy include depression as well as swollen, bleeding gums and loosening or loss of teeth due to tissue and capillary fragility [ 6 , 8 , 9 ].
Iron deficiency anemia can also occur due to increased bleeding and decreased nonheme iron absorption secondary to low vitamin C intake [ 6 , 11 ]. In children, bone disease can be present [ 6 ]. Left untreated, scurvy is fatal [ 6 , 9 ]. Until the end of the 18 th century, many sailors who ventured on long ocean voyages, with little or no vitamin C intake, contracted or died from scurvy.
During the mids, Sir James Lind, a British Navy surgeon, conducted experiments and determined that eating citrus fruits or juices could cure scurvy, although scientists did not prove that ascorbic acid was the active component until [ ]. Today, vitamin C deficiency and scurvy are rare in developed countries [ 8 ].
Vitamin C deficiency is uncommon in developed countries but can still occur in people with limited food variety. The following groups are more likely than others to be at risk of obtaining insufficient amounts of vitamin C. Studies consistently show that smokers have lower plasma and leukocyte vitamin C levels than nonsmokers, due in part to increased oxidative stress [ 8 ].
Office Heat – Erotic Short Story for Women ebook by Rose Boyd
For this reason, the IOM concluded that smokers need 35 mg more vitamin C per day than nonsmokers [ 8 ]. Exposure to secondhand smoke also decreases vitamin C levels. Although the IOM was unable to establish a specific vitamin C requirement for nonsmokers who are regularly exposed to secondhand smoke, these individuals should ensure that they meet the RDA for vitamin C [ 4 , 8 ].
For many reasons, feeding infants evaporated or boiled cow's milk is not recommended. This practice can cause vitamin C deficiency because cow's milk naturally has very little vitamin C and heat can destroy vitamin C [ 6 , 12 ]. Although fruits and vegetables are the best sources of vitamin C, many other foods have small amounts of this nutrient.
Thus, through a varied diet, most people should be able to meet the vitamin C RDA or at least obtain enough to prevent scurvy. People who have limited food variety—including some elderly, indigent individuals who prepare their own food; people who abuse alcohol or drugs; food faddists; people with mental illness; and, occasionally, children—might not obtain sufficient vitamin C [ 4 , , 11 ].
People with severe intestinal malabsorption or cachexia and some cancer patients might be at increased risk of vitamin C inadequacy [ 29 ]. Low vitamin C concentrations can also occur in patients with end-stage renal disease on chronic hemodialysis [ 30 ]. This section focuses on four diseases and disorders in which vitamin C might play a role: cancer including prevention and treatment , cardiovascular disease, age-related macular degeneration AMD and cataracts, and the common cold.
Epidemiologic evidence suggests that higher consumption of fruits and vegetables is associated with lower risk of most types of cancer, perhaps, in part, due to their high vitamin C content [ 1 , 2 ].
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Vitamin C can limit the formation of carcinogens, such as nitrosamines [ 2 , 31 ], in vivo; modulate immune response [ 2 , 4 ]; and, through its antioxidant function, possibly attenuate oxidative damage that can lead to cancer [ 1 ]. Most case-control studies have found an inverse association between dietary vitamin C intake and cancers of the lung, breast, colon or rectum, stomach, oral cavity, larynx or pharynx, and esophagus [ 2 , 4 ].
Plasma concentrations of vitamin C are also lower in people with cancer than controls [ 2 ]. However, evidence from prospective cohort studies is inconsistent, possibly due to varying intakes of vitamin C among studies. Evidence from most randomized clinical trials suggests that vitamin C supplementation, usually in combination with other micronutrients, does not affect cancer risk.
MAX study, a randomized, double-blind, placebo-controlled clinical trial,13, healthy French adults received antioxidant supplementation with mg ascorbic acid, 30 mg vitamin E, 6 mg beta-carotene, mcg selenium, and 20 mg zinc, or placebo [ 35 ]. After a median follow-up time of 7. In addition, baseline antioxidant status was related to cancer risk in men, but not in women [ 36 ].
Similar findings were reported in women participating in the Women's Antioxidant Cardiovascular Study [ 38 ]. In a large intervention trial conducted in Linxian, China, daily supplements of vitamin C mg plus molybdenum 30 mcg for 5—6 years did not significantly affect the risk of developing esophageal or gastric cancer [ 39 ]. Moreover, during 10 years of follow-up, this supplementation regimen failed to significantly affect total morbidity or mortality from esophageal, gastric, or other cancers [ 40 ].
A review of vitamin C and other antioxidant supplements for the prevention of gastrointestinal cancers found no convincing evidence that vitamin C or beta-carotene, vitamin A, or vitamin E prevents gastrointestinal cancers [ 41 ]. A similar review by Coulter and colleagues found that vitamin C supplementation, in combination with vitamin E, had no significant effect on death risk due to cancer in healthy individuals [ 42 ].
At this time, the evidence is inconsistent on whether dietary vitamin C intake affects cancer risk. Results from most clinical trials suggest that modest vitamin C supplementation alone or with other nutrients offers no benefit in the prevention of cancer. A substantial limitation in interpreting many of these studies is that investigators did not measure vitamin C concentrations before or after supplementation.
Plasma and tissue concentrations of vitamin C are tightly controlled in humans. At daily intakes of mg or higher, cells appear to be saturated and at intakes of at least mg, plasma concentrations increase only marginally [ 2 , 10 , 24 , 33 , 39 ]. If subjects' vitamin C levels were already close to saturation at study entry, supplementation would be expected to have made little or no difference on measured outcomes [ 24 , 25 , 43 , 44 ].